For Physical Therapist Clinicians
The resources here are intended for use by qualified and licensed healthcare providers, who take full responsibility for how they use these resources. Nothing here should replace care from a qualified medical provider.
Clinical Considerations: A Guide for Physical Therapists
Treating patients with Ehlers-Danlos syndrome (EDS) requires rethinking many standard physical therapy assumptions. These patients are not simply deconditioned or fearful—they present with altered connective tissue mechanics, impaired proprioception, and nervous system adaptations shaped by repeated injury and instability [Castori 2012; Malfait 2017].
Effective care depends as much on how therapy is delivered as what exercises are prescribed.
Psychosocial Factors Strongly Influence Outcomes
Most patients with EDS enter physical therapy with a complex psychosocial background that directly affects participation and progress.
Common features include:
Kinesiophobia grounded in lived injury experience rather than avoidance behavior [Castori 2012]
Prior harm in PT, often due to overly aggressive strengthening or stretching [Celletti 2015]
Medical invalidation or gaslighting, particularly when imaging is normal despite severe symptoms [Castori 2017]
Hopelessness or catastrophizing (“this will never get better”) [Clauw 2014]
Difficulty identifying triggers, especially with delayed symptom flares [Engelbert 2017]
High comorbidity burden, including dysautonomia/POTS, GI dysfunction, migraines, pelvic floor dysfunction, and MCAS [Eccles 2012; Tinkle 2017]
These patients are often labeled as “fragile,” when in reality they are highly adaptive, skilled compensators operating in a system with poor passive stability.
Validation, predictability, and collaborative pacing are therapeutic interventions.
Session Flow Must Account for Autonomic Dysfunction
Autonomic symptoms are common and clinically relevant in EDS [Eccles 2012; Mathias 2021].
Clinical implications:
Avoid rapid position changes
Consider starting sessions supine or seated
Build rest breaks into sessions proactively
Monitor vitals as appropriate (HR, BP, symptom response)
Recognize that orthostatic stress can impair motor learning
Symptom exacerbation related to autonomic load should not be misinterpreted as lack of effort or poor tolerance.
Continuous Feedback Is Essential
Patients with EDS often cannot accurately assess symptom response in real time due to:
impaired proprioception
delayed-onset pain
nervous system sensitization [Rombaut 2015; Woolf 2011]
Best practices include:
Requesting ongoing feedback during sessions
Tracking later-day and next-day responses
Normalizing exercise modification or discontinuation
Avoiding language that frames adjustment as “regression”
Delayed flares are data—not noncompliance.
FORM IS THE PRIMARY DOSING VARIABLE
Form and joint control matter more than intensity, volume, or repetition count.
Before strengthening:
Assess for excessive global holding and guarding
Teach downregulation of resting muscle tone
Reduce reliance on secondary stabilizers
Strength layered onto chronic guarding increases pain, fatigue, and inefficiency [Celletti 2015; Engelbert 2017].
Strengthening Principles Specific to EDS
Core Comes First—but With Redefined Goals
Core training should prioritize:
alignment and midline control
subtle activation rather than maximal effort
integration with breathing
transfer to functional movement over time
Traditional core paradigms (planks, crunches, long-lever challenges) are often poorly tolerated early [Celletti 2015].
Favor Isometrics and Closed-Chain Work
These approaches:
minimize joint shear
enhance proprioceptive input
support mid-range control
reduce reliance on end-range stability [Panagiotopoulou 2016; Rombaut 2015]
Progressions should remain submaximal for longer than in non-hypermobile populations.
Mid-Range, Low-Impact, Submaximal Loading
Key considerations:
Avoid end-range loading
Avoid high-impact or ballistic tasks
Delay heavy push/pull/lift
Accept that sessions may not be pain-free, but must avoid post-exertional symptom flares that last into the next day
Hyperalgesic nervous systems require conservative loading strategies [Clauw 2014; Woolf 2011].
Proprioception Is a Core Impairment
Proprioceptive deficits are well-documented in EDS and hypermobility-related disorders [Rombaut 2015; Scheper 2016].
Assessment should include:
ability to find and return to neutral
control with reduced visual input
movement quality under low load
joint drift, locking, or excessive co-contraction
Do not assume proprioception will normalize as a byproduct of strengthening—treat it explicitly [Engelbert 2017].
Address Muscle Tone in Two Distinct Domains
EDS patients require intervention in two related but separate areas:
1. Static Resting Muscle Tone
Goals:
reduce baseline guarding
decrease overactivity of secondary stabilizers
improve comfort and fatigue levels
2. Dynamic Muscle Tone and Endurance
Goals:
support functional joint stability
improve task-specific control
increase tolerance for daily activities
Progressing dynamic strength without addressing static overactivity increases symptom burden [Celletti 2015; Castori 2017].
Redefining Progress in patients with EDS
Progress may present as:
fewer or shorter flares
faster recovery after activity
reduced guarding
improved confidence in movement
improved tolerance rather than peak output
Improvement in tolerance for ADLs
Linear progression is uncommon. Sustainable progress is the clinical goal [Engelbert 2017].
Clinical Bottom Line
Patients with EDS are not fragile—they are biomechanically and neurologically distinct.
Effective physical therapy prioritizes:
control over range
precision over intensity
consistency over volume
collaboration over compliance
When therapy aligns with tissue behavior and nervous system needs, outcomes improve and trust is restored.